By N. W. März, M. Nauck, H. Wieland (auth.), Dr. A. Geibel, Prof. Dr. H. Just, Dr. med. W. Kasper, Dr. S. Konstantinides (eds.)
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Extra info for Acute Pulmonary Embolism: A Challenge for Hemostasiology
Thromb Haemost: p 337; PS-1384 24. Dahlbiick B (1986) Inhibition of protein Ca cofactor function of human and bovine protein S by C4bbinding protein. J Bioi Chern 261: 12022-12027 25. Dahlbiick B (1995) Inherited thrombophilia: Resistance to activated protein C as a pathogenic factor of venous thromboembolism. Blood 85: 607-614 26. Dahlbiick B, Carlsson M, Svensson PJ (1993) Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: Prediction of a cofactor to activated protein C.
Yasuda M, Takeuchi K, Hiruma M, Iida H, Tahara A, Itagane H, Toda I, Akioka K, Teragaki M, Oku H, Kanayama Y, Takeda T, Kolb WP, Tamerius JD (1990) The complement system in ischemic heart disease. Circulation 81: 156-163 26. Zalewski A, Shi Y, Nardone D, Bravette B, Weinstock P, Fischmann D, Wilson P, Goldberg S, Levin DC, Bjornsson TD (1991) Evidence for reduced fibrinolytic activity in unstable angina at rest. Circulation 83: 1685-1691 Prof. Dr. Hans Martin Hoffmeister (181) . W. Heller· 1. Seipel Medizinische Universitatsklinik Abteilung Innere Medizin III Otfried-Miiller-StraBe 10 72076 Tiibingen, Germany 27 Clinical implications of the new understanding of thrombophilia S.
5 % were carriers of factor V Leiden. After a median observation time of 19 months the patients with factor V Leiden had the same recurrence rate as the patient group without factor V Leiden . Antiphospholipid antibodies Antiphospholipid antibodies are acquired antibodies directed against phospholipids or phospholipid-binding proteins . They are risk factors for venous [43,56,94,103] and arterial [1,57,68] thrombosis. The term "lupus anticoagulant" refers to the subgroup of antiphospholipid antibodies in which one or several of the coagulation screening tests (prothrombin time, activated prothrombin time, dilute Russell viper venom time) are prolonged and where the prolongation can be overcome by the addition of extra phospholipids.
Acute Pulmonary Embolism: A Challenge for Hemostasiology by N. W. März, M. Nauck, H. Wieland (auth.), Dr. A. Geibel, Prof. Dr. H. Just, Dr. med. W. Kasper, Dr. S. Konstantinides (eds.)