Get Advances in Biomedical Engineering. Volume 3 PDF

By J.H.U. Brown, J.F. Dickson

ISBN-10: 0120049031

ISBN-13: 9780120049035

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Permutt et al. (1969) presented a bulk of experimental evidence that the pulmonary veins and microvessels are almost indistensible, thus supporting the first of the two mechanisms as the only source for the nonlinear pulmonary resistance. Karp et al. (1968) reported a similar finding. In contrast, Glazier et al. (1969) showed in quickly frozen lungs perfused with various arterial and venous pressures that the capillary diameter was similar in the lower lobes but varied in the middle lobes with perfusion pressures.

Levy et al. (1963) showed that, when the temporary elevation of end-diastolic ventricular pressure was prevented after a sudden increase of aortic pressure load, the extent of homeometric autoregulation judged by the recovery of stroke volume and work toward the control was considerably diminished compared with the case in which the end-diastolic pressure was allowed to rise temporarily. Clancy et al. (1968) reported that the end-diastolic circumference, measured by a mercury gauge, tended to increase despite the seemingly identical end-diastolic pressure following the apparent aortic pressure-induced homeometric autoregulation.

31). However, inertance of blood was neglected in Modell's model. Therefore, the following equations described the behavior of an nth compartment (n = pa, ea, alv, or pv). Blood volume : Qn = Qon + f(Qn-i — Qn) dt Pressure : Pn_x - Pn = QnRn Pn - = PEn = ^ Ptm (Qon = initial blood volume) (54) (55) J(Qn - (56) Qon) dt PEU represents extramural pressure, Ptm transmural pressure, and Q0n initial flow for the nth compartment. i. Pulmonary artery. 0030 mm Hg per milliliter per second. 7 mm Hg. 1 ml/mm Hg, which is about Qn-I P n _, "^ O — WW Qn Pn Rn Cn FIG.

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Advances in Biomedical Engineering. Volume 3 by J.H.U. Brown, J.F. Dickson

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